Stub Mandrel

Ah the benefits of a private healthcare system... no-one can afford to be ill.

Dangnabbit. One of the few instruments to have a tune written to celebrate it.

I think the point is to show what it would look like with a more conventional headstock. An option could be buying one and and chopping refinishing the headstock.

You will learn faster the more you mix it up. Also worth playing arpeggios and randomly noodling within a scale - especially if you change positions.

I have a suspicion that the Coronavirus was genetically engineered by Millennial and Gen-Xers specifically to target Baby Boomers... AGE DEATH RATE all cases 80+ years old 14.8% 70-79 years old 8.0% 60-69 years old 3.6% 50-59 years old 1.3% 40-49 years old 0.4% 30-39 years old 0.2% 20-29 years old 0.2% 10-19 years old 0.2% 0-9 years old no fatalities

Consider fitting 5mm x 3mm neodymium magnets, you have to push the pole pieces down into the body. Turns Squier PUPpies into ravaging wolves 🙂

"Bite" me off a piece of that 🙂

John Entwhistle too, he whacks the strings down against the frets with his fingertips, playing over the end of the fretboard, something I like doing when I want an aggressive sound.

Day Tripper. Only ever learnt the main riff before. Quite subtle and very inventive.

It's based on a figure of 50% of people catching it worldwide, so it's unlikely to be an under-estimate.

Both, although the better I get at bass the more I realise how bad my guitar playing is.

Without any ill-will to Phil Collins, I'd only go and see Genesis if they replaced him with Peter Gabriel and did the early stuff instead of the pop/AOR.

It's supposedly derived from the Gibson EB-1 although it looks more like a Hofner with the horrible rectangular control plate.

I may be wrong, but I think the 'Clive Button' version was the four-mosfet power amp design as used in the keyboard amps which is relatively bullet proof compared to the bipolar design.

I bought some nice 20dB ones but they were far too effective. For Christmas my daughter asked for a pair of V-moda faders (much loved by techno and D&B lovers, apparently) which are supposedly 12dB so I got myself a pair. Reduced from £29 to £19 at Bax. They do seem to keep clarity and I think the lower attenuation is more practical. https://www.v-moda.com/us/en/products/faders

My first electric guitar was this KT-2 bought second hand in 1980 (about the time Running Free came out...) It has the same PUPS and I can confirm that they are microphonic (well they would be, the coils just lie loose in the pickup body). It actually sounds pretty 'vintage' and feeds back like a monster... (I fitted the tailpiece and tune-a-matic to replace the plain bar bridge and sub-sub-bigsby trem. And knobs and switches)

? Surely the original thread is a valuable documenting of the build? (BTW you could have changed the original title by editing your first post in the thread).

Still they appear to've lost the other basses and >gasp< the gorgeous Masterbilt guitars 😥

The obvious route is to temporarily plug the holes in the bridge pieces before ageing them, then fit un-corroded grub screws. This appears to be how Fender did it on my relic bass.

Not according to some sources: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3029258/ The “Spanish” Influenza Pandemic of 1918–1919 In early March of 1918, several severe cases of influenza were reported in the United States. This would mark the beginning of the first wave of the “Spanish” influenza pandemic (reviewed in [1]). As massive numbers of US military troops were deployed in Europe, the virus spread too, leading to outbreaks throughout the United States, Europe, and possibly Asia. Spain was neutral during the war and its press was therefore uncensored. Accordingly, the Spanish newspapers were filled with reports of the disease, especially when the king became ill. It is believed that these published accounts of the spread of the disease explain why the virus became known as the “Spanish” influenza. During the first wave of this, like other, pandemics, mortality rates were in the normal range, though morbidity rates were high. However, while the first wave killed relatively few, by the time the next wave came, in the fall of 1918, the virus had undergone numerous passages through humans, had changed dramatically, and was now lethal. Together with a third wave, which occurred in the winter of 1918–1919, approximately 30% of the world's population (500 million people) is thought to have been clinically affected by the 1918 pandemic [1]. Unexpectedly, the 1918 pandemic attacked particularly young adults, who usually have a low death rate during influenza epidemics. As a result, influenza and pneumonia death rates for 15- to 34-year-olds were more than 20 times higher in 1918 than in the previous year (the mortality rate associated with the 1918 virus was more than 2.5% among infected persons compared with less than 0.1% in other influenza epidemics; [1]). Most deaths were caused from secondary bacterial pneumonia due to a lack of antibiotics [1]. The 1918 pandemic virus, however, also killed quickly and directly with a violent viral pneumonia, often with either massive acute pulmonary hemorrhage or pulmonary edema. The disease course was frequently less than 5 days [1]. ::: Although the complete sequences of the viral RNAs of the 1918 pandemic virus have been determined, the viral genome does not contain any motifs known to be associated with high virulence [5]. Therefore, to understand the extraordinary virulence of the 1918 pandemic virus, it was important to re-create the virus and examine its pathogenicity in animals. The recent technological advancement of reverse genetics, which allows the generation of infectious influenza viruses entirely from cDNAs [11], made possible the re-creation of the 1918 pandemic virus. Tumpery's group at the US Centers for Disease Control and Prevention [12] and we [13] succeeded in rescuing viruses bearing all eight RNA segments of the 1918 virus by using reverse genetics. Now that we had all of the materials required, we could study the molecular properties associated with the unusual virulence of the 1918 pandemic virus. ::: Which viral factor(s) contribute to the extraordinary virulence of the 1918 virus? One of the key players is hemagglutinin (HA), which is the viral surface glycoprotein that has two functions in the early stage of virus replication: receptor binding and membrane fusion. The interesting thing being that, unusually for flu, it killed through viral pneumonia, the same way as Covid-19.

Than normal flu. The 1918 strain was far more lethal than ordinary flu.

The 2018 flu pandemic killed a quarter of a million people in the UK. It's calculated that 50% of the population was infected. The death rate was probably around 2-3%. A the time one could equally have said: " The first thing to note is that the vast majority of influenza cases produce symptoms similar to a cold. Of those that are more serious than a cold, the vast majority produce symptoms similar to flu. Of those that are more serious than flu, most sufferers develop very nasty symptoms that can be life-threatening to a minority of the population. And finally you reach the cases that the media are drooling over ... people dying. " The main difference is that it appeared to have an additional peak in mortality for 30-year olds: Coronavirus seems to be just as infectious, but may have a lower death rate - we really don't know as there simply aren't reliable figures on how many people are infected without symptoms (or who hide them). The things that are certain are that we still have too little data to really understand how the outbreak will progress; it could be a year or more before we have a vaccine to enable us to do more than we could have done for historic pandemics; and if this one does fizzle out, there will eventually be one that doesn't.

Crikey Ped, when did you last change those strings? Hourglass wear at the 20h fret?!

Mysteries of the universe... eBay (and the wider shopping world) is awash with cheap Jazz bass pickup covers, J-bridge covers and P-bridge covers. But you can only get P-bass pickup covers sourced from Fender. Does anyone know why?